Hi All,
I am not a geneticist so my understanding of matters such as DNA and chromosomal abnormalities like the Philadelphia Chromosome that we all suffer ,is limited.
However I am keen to find out if it might be at all possible to repair my DNA which has been compromised via vaccines or perhaps diet and lifestyle maybe.
I came across an article in a magazine that explained that telomeres are structures found at the ends of chromosomes and provide protection during cell division ;telomere length is important so like a clock the shorter the telomere the older the cell and a diet high in inflammatory foods speeds up the aging process.
We are often told about the nasties in foods that act as inflammatories such as added sugar and especially high-fructose corn syrup found in beverages and pastries for example .There are trans fats in fried food ,processed snacks and margarine ;add in processed meats refined carbohydrates such as white flour and white rice .Excess omega -6 is found in mayonnaise salad dressings and vegetable oils like corn safflower and soy.
On the other hand foods that reduce inflammation are fruits and vegetables of strong colour ,olive oil ,omega 3 rich fish plus nuts .
Having read books on lifestyle after cancer or living with cancer I wonder to what extent we can maintain good chromosome health especially in our cases where we suffer the abnormality of the Ph+condition? Some sufferers of other cancers such as breast cancer and thyroid cancer for instance have taken on near vegan diets and focus as well on exercise ,adequate sleep and the management of stress during and post treatment and consider that this helps to stave off disease progression??
Has anyone else gone down the route of modification of diet and lifestyle in the hope that it might assist with disease management?
I am not advocating what some have suggested say for instance that high dose resveratol (found in black grape skins) as a replacement for tkis and other alternative therapies ;nor am I being prescriptive on diet etc .
I can find very little published on the relation between telomere length and CML except one or two short academic articles that conclude:
"A marked reduction in telomere length is associated with disease progression in CML"
"Telomere shortening is a well known marker of aging and cellular stress"
Regards
John
